What is it about?
Our findings show that the increased thrombotic risk associated with high blood pressure is mediated by a mechanism that is dependent on both T cells (a subtype of white blood cell) and interleukin-6 (IL-6).
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Why is it important?
Drug discovery programs based on T cell dependent IL-6 signaling pathways may provide a previously unknown therapeutic strategy for the management of both thrombotic and inflammatory complications that are associated with people with high blood pressure (hypertension).
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This page is a summary of: A critical role for both CD40 and VLA5 in angiotensin II–mediated thrombosis and inflammation, The FASEB Journal, June 2018, Federation of American Societies For Experimental Biology (FASEB),
DOI: 10.1096/fj.201701068r.
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