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  1. Anthocyanins in cardioprotection: A path through mitochondria
  2. Effects of sevoflurane vs. propofol on mitochondrial functional activity after ischemia-reperfusion injury and the influence on clinical parameters in patients undergoing CABG surgery with cardiopulmonary bypass
  3. Anthocyanins as substrates for mitochondrial complex I - protective effect against heart ischemic injury
  4. Neuroprotective effects of nitric oxide donor NOC-18 against brain ischemia-induced mitochondrial damages: role of PKG and PKC
  5. Effects of standardized extract of Ginkgo biloba leaves EGb761 on mitochondrial functions: mechanism(s) of action and dependence on the source of mitochondria and respiratory substrate
  6. Small Aβ1-42oligomer-induced membrane depolarization of neuronal and microglial cells: Role of N-methyl-D-aspartate receptors
  7. Microcirculatory, mitochondrial, and histological changes following cerebral ischemia in swine
  8. Influence of NADPH oxidase on inflammatory response in primary intestinal epithelial cells in patients with ulcerative colitis
  9. Antibodies bound to Aβ oligomers potentiate the neurotoxicity of Aβ by activating microglia
  10. In the eye of the storm: mitochondrial damage during heart and brain ischaemia
  11. Experimental acute pancreatitis induces mitochondrial dysfunction in rat pancreas, kidney and lungs but not in liver
  12. Experimental acute pancreatitis induces mitochondrial dysfunction in rat pancreas, kidney and lungs but not in liver
  13. P053 Influence of NADPH oxidase on inflammatory response in the primary intestinal epithelial cells from patients with ulcerative colitis
  14. Anthocyanins block ischemia-induced apoptosis in the perfused heart and support mitochondrial respiration potentially by reducing cytosolic cytochrome c
  15. Immunogenic properties of amyloid beta oligomers
  16. Mitochondrial involvement in regulation of apoptosis via redox state of external cytochrome c
  17. Influence of Ethanol Extract of Ginkgo biloba Leaves on the Isolated Rat Heart Work and Mitochondria Functions
  18. Molecular, histological and microcirculatory modeling of cerebral ischemia in pigs
  19. There is no evidence that mitochondria are the main source of reactive oxygen species in mammalian cells
  20. Phenomenological Kinetic and Control Analysis of Oxidative Phosphorylation in Isolated Mitochondria
  21. Beta-amyloid oligomers: recent developments
  22. Tetramethylphenylenediamine protects the isolated heart against ischaemia-induced apoptosis and reperfusion-induced necrosis
  23. Mitochondria in regulation of cell death in cardiovascular and brain pathologies
  24. Estradiol-induced protection against ischemia-induced heart mitochondrial damage and caspase activation is mediated by protein kinase G
  25. Size-dependent neurotoxicity of β-amyloid oligomers
  26. Effects of Ginkgo biloba extract on heart and liver mitochondrial functions: mechanism(s) of action
  27. Mitochondria as decision-makers in cell death
  28. Effects of ischemia-reperfusion and pretreatment with mildronate on rat liver mitochondrial function
  29. Nitric oxide protects the heart from ischemia-induced apoptosis and mitochondrial damage via protein kinase G mediated blockage of permeability transition and cytochrome c release
  30. Regulation of apoptosis by the redox state of cytochrome c
  31. S12.35 Mitochondria as regulators of apoptosis through the redox state of cytochrome c
  32. AMPK, MAPK and Bax in the heart: some questions answered
  33. Mitochondrial Regulation of Caspase Activation by Cytochrome Oxidase and Tetramethylphenylenediamine via Cytosolic Cytochrome c Redox State
  34. Nitric oxide and mitochondrial respiration in the heart
  35. Nitric oxide from neuronal nitric oxide synthase sensitises neurons to hypoxia-induced death via competitive inhibition of cytochrome oxidase
  36. The cardioprotective effect of NOC-18 on ischemia/reperfusion induced mitochondrial damage and cell viability
  37. Interactions between nitric oxide, oxygen, reactive oxygen species and reactive nitrogen species
  38. Estradiol prevents release of cytochrome c from mitochondria and inhibits ischemia-induced apoptosis in perfused heart
  39. Estradiol prevents release of cytochrome c from heart mitochondria after ischemia due to activation of estrogen receptors
  40. NO and estrogens protect against ischaemia-induced apoptosis by inhibiting mitochondrial permeability transition
  41. S-nitrosothiol inhibition of mitochondrial complex I causes a reversible increase in mitochondrial hydrogen peroxide production
  42. What else has to happen for nitric oxide to induce cell death?
  43. What else has to happen for nitric oxide to induce cell death?
  44. NITRIC OXIDE FROM INDUCIBLE NITRIC OXIDE SYNTHASE SENSITIZES THE INFLAMED AORTA TO HYPOXIC DAMAGE VIA RESPIRATORY INHIBITION
  45. Inhibition of mitochondrial respiratory complex I by nitric oxide, peroxynitrite and S-nitrosothiols
  46. Nitric oxide from inflammatory-activated glia synergizes with hypoxia to induce neuronal death
  47. Nitric oxide induces apoptosis via hydrogen peroxide, but necrosis via energy and thiol depletion
  48. Nitric oxide induces apoptosis via hydrogen peroxide, but necrosis via energy and thiol depletion
  49. S-Nitrosothiol-induced rapid cytochrome c release, caspase activation and mitochondrial permeability transition in perfused heart
  50. Inhibition of mitochondrial permeability transition prevents mitochondrial dysfunction, cytochrome c release and apoptosis induced by heart ischemia
  51. Mitochondria in apoptosis of ischemic heart
  52. Nitric oxide inhibition of mitochondrial respiration and its role in cell death
  53. Inhibition of mitochondrial permeability transition and release of cytochrome c by anti-apoptotic nucleoside analogues
  54. Nitric oxide and hypoxia target cytochrome oxidase to induce apoptosis and necrosis via mitochondrial permeability transition
  55. Nitrosation causes mitochondrial dysfunction and cytochrome c release in myocardium
  56. Estrogens prevent calcium-induced release of cytochrome c from heart mitochondria
  57. Mitochondrial disappearance from cells: a clue to the role of autophagy in programmed cell death and disease?
  58. Nitric Oxide, Mitochondria, and Cell Death
  59. Release of mitochondrial cytochrome c and activation of cytosolic caspases induced by myocardial ischaemia
  60. Caspases are reversibly inactivated by hydrogen peroxide
  61. Reversal of nitric oxide-, peroxynitrite- and S-nitrosothiol-induced inhibition of mitochondrial respiration or complex I activity by light and thiols
  62. Nitric oxide donors, nitrosothiols and mitochondrial respiration inhibitors induce caspase activation by different mechanisms
  63. Mitochondria Mediate Nitric Oxide-Induced Cell Death
  64. Superoxide dismutase and hydrogen peroxide cause rapid nitric oxide breakdown, peroxynitrite production and subsequent cell death
  65. Release of cytochrome c from heart mitochondria is induced by high Ca2+ and peroxynitrite and is responsible for Ca2+-induced inhibition of substrate oxidation
  66. 12 Cytochrome c and activation of caspases during myocardial ischaemia
  67. Kinetic Analysis of Changes in Activity of Heart Mitochondrial Oxidative Phosphorylation System Induced by Ischemia
  68. Control and kinetic analysis of ischemia-damaged heart mitochondria: which parts of the oxidative phosphorylation system are affected by ischemia?
  69. The function of ATP/ADP translocator in the regulation of mitochondrial respiration during development of heart ischemic injury
  70. Control of Respiration in Heart Mitochondria: Comparative Study of Oxidation of Succinate and NAD-Dependent Substrates
  71. Development of the technique and methods of myoventriculoplasty with use of programmable cardiosynchronized electroneurostimulation
  72. Role of an adenine-nucleotide translocator in regulation of mitochondrial pyruvate oxidation in the heart