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  1. Prolactin/Stat5 and Androgen R1881 Coactivate Carboxypeptidase-D Gene in Breast Cancer Cells
  2. Carboxypeptidase-D is elevated in prostate cancer and its anti-apoptotic activity is abolished by combined androgen and prolactin receptor targeting
  3. Progestin-inducible EDD E3 ubiquitin ligase binds to α4 phosphoprotein to regulate ubiquitination and degradation of protein phosphatase PP2Ac
  4. Abstract 86: Prolactin and testosterone induction of carboxypeptidase-D to promote cell survival is greater in prostate cancer cells than benign prostate cells, and their synergistic action in prostate cancer cells is effectively blocked by receptor an...
  5. Testosterone and prolactin increase carboxypeptidase-D and nitric oxide levels to promote survival of prostate cancer cells
  6. Overexpression of 5α-reductase type 1 increases sensitivity of prostate cancer cells to low concentrations of testosterone
  7. Effect of Steroid 5α-Reductase Inhibitors on Markers of Tumor Regression and Proliferation in Prostate Cancer
  8. Type 1 and Type 2 5α-Reductase Expression in the Development and Progression of Prostate Cancer
  9. Levels of 5α-Reductase Type 1 and Type 2 are Increased in Localized High Grade Compared to Low Grade Prostate Cancer
  10. Fetal Distribution of 5α-Reductase 1 and 5α-Reductase 2, and Their Input on Human Prostate Development
  11. The effects of the dual 5α-reductase inhibitor dutasteride on localized prostate cancer—results from a 4-month pre-radical prostatectomy study
  12. RE: EFFECT OF THE DUAL 5α-REDUCTASE INHIBITOR DUTASTERIDE ON MARKERS OF TUMOR REGRESSION IN PROSTATE CANCER
  13. Differential alterations in 5?-reductase type 1 and type 2 levels during development and progression of prostate cancer
  14. EFFECT OF THE DUAL 5α-REDUCTASE INHIBITOR DUTASTERIDE ON MARKERS OF TUMOR REGRESSION IN PROSTATE CANCER
  15. Dutasteride, the dual 5?-reductase inhibitor, inhibits androgen action and promotes cell death in the LNCaP prostate cancer cell line
  16. 5α-Reductase Type 1 Immunostaining is Enhanced in Some Prostate Cancers Compared With Benign Prostatic Hyperplasia Epithelium
  17. Distribution and activity of 5 alpha-reductase isoenzymes in benign prostatic hyperplasia and prostate cancer
  18. Prostatic involution in men taking finasteride is associated with elevated levels of insulin-like growth factor-binding proteins (IGFBPs)-2, -4, and -5
  19. Prostatic involution in men taking finasteride is associated with elevated levels of insulin-like growth factor-binding proteins (IGFBPs)-2, -4, and -5
  20. THE UTILITY OF TISSUE TRANSGLUTAMINASE AS A MARKER OF APOPTOSIS DURING TREATMENT AND PROGRESSION OF PROSTATE CANCER
  21. Androgen-Induced Regrowth in the Castrated Rat Ventral Prostate: Role of 5α-Reductase1
  22. Insulin-like growth factor binding protein 5 is associated with involution of the ventral prostate in castrated and finasteride-treated rats
  23. Insulin‐like growth factor binding protein 5 is associated with involution of the ventral prostate in castrated and finasteride‐treated rats
  24. Relative potency of testosterone and dihydrotestosterone in preventing atrophy and apoptosis in the prostate of the castrated rat.
  25. Apoptosis is the mode of keratinocyte death in cutaneous graft-versus-host disease
  26. Evidence for atrophy and apoptosis in the prostates of men given finasteride.
  27. Evidence for atrophy and apoptosis in the ventral prostate of rats given the 5 alpha-reductase inhibitor finasteride.