What is it about?
The present study investigated how voluntary exercise modulated insulin-stimulated glucose transport and the protein expression and phosphorylation status of the signaling molecules in the glucose transport process in skeletal muscle as well as other cardiometabolic risks in a rat model with insulin resistance syndrome induced by chronic angiotensin II (ANGII) infusion.
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Why is it important?
We found that voluntary exercise is an intervention that can alleviate insulin resistance of glucose transport and impaired insulin signaling molecules in skeletal muscle and improve whole-body insulin sensitivity in rats chronically administered with ANGII.
Perspectives
Our findings underscore the benefits of an active lifestyle for the prevention of cardiometabolic risks that might be developed due to elevated levels of ANGII.
Vitoon S
Mahidol University
Read the Original
This page is a summary of: Exercise Protects Against Defective Insulin Signaling and Insulin Resistance of Glucose Transport in Skeletal Muscle of Angiotensin II-Infused Rat, Frontiers in Physiology, April 2018, Frontiers,
DOI: 10.3389/fphys.2018.00358.
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