What is it about?
The early normal activities of the maternal thyroid gland may be a regulator of the normal placentation. In addition, maternal thyroid dysfunctions (hyperthyroidism or hypothyroidism) can impair the placental growth factors, cytokines, and blood supply. The placental dysfunction may increase the risk of preeclampsia, fetal growth restriction and mortality. Thus, treatment of thyroid disorders before or during the gestation may decrease the risk of pregnancy complications. Additional investigations are necessary not only to examine the biological and molecular mechanisms between the thyroid dysfunction and placental disorders, preeclampsia and premature delivery but also to compare the data with the clinical studies.
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Why is it important?
he placental dysfunction may increase the risk of preeclampsia, fetal growth restriction and mortality. T
Perspectives
Additional investigations are necessary not only to examine the biological and molecular mechanisms between the thyroid dysfunction and placental disorders, preeclampsia and premature delivery but also to compare the data with the clinical studies.
Full Professor Ahmed R. G.
Division of Anatomy and Embryology, Zoology department, Faculty of Science, Beni-Suef University, Egypt.
Read the Original
This page is a summary of: Maternal Thyroid Function and Placental Hemodynamics, ARC Journal of Animal and Veterinary Sciences, January 2018, ARC Publications Pvt Ltd.,
DOI: 10.20431/2455-2518.0401002.
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