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Different hypotheses have been presented to explain the cellular mechanism for chronic kidney disease due to variant forms of Apolipoprotein-L1 (APOL1). In this Perspective, I propose a model accounting for most observations, and explaining the main differences between models.

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This page is a summary of: The Mechanism of Kidney Disease due to APOL1 Risk Variants, Journal of the American Society of Nephrology, April 2024, Wolters Kluwer Health,
DOI: 10.1681/asn.0000000000000367.
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