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The host protease TMPRSS2 plays an essential role for activation of influenza viruses. However, influenza virus becomes able to use another protease after the loss of a sugar at the hemagglutinin stalk region.
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This page is a summary of: A Mutant H3N2 Influenza Virus Uses an Alternative Activation Mechanism in TMPRSS2 Knockout Mice by Loss of an Oligosaccharide in the Hemagglutinin Stalk Region, Journal of Virology, February 2015, ASM Journals,
DOI: 10.1128/jvi.00124-15.
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