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The DNA helicase Fbh1 serves a vital meiotic role in promoting normal chromosome segregation. In the absence of Fbh1, chromosomes remain entangled or segregate unevenly during meiosis; we show that this is caused by a failure to efficiently dismantle Rad51 nucleofilaments that form during meiotic double-strand break repair.
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This page is a summary of: A failure of meiotic chromosome segregation in a fbh1 mutant correlates with persistent Rad51-DNA associations, Nucleic Acids Research, December 2010, Oxford University Press (OUP),
DOI: 10.1093/nar/gkq977.
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