Wireless radiation affects insulin activity & sensitivity and causes adverse biological effects

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Why is it important?

The effects discussed in this diabetes case study have tremendous public health implications. Radiation from wireless technology is now a ubiquitous exposure, so is "dirty" electricity. The incidences of diabetes and pre-diabetes are increasing. This case suggests that the increases in ambient wireless radiation and "dirty" electricity levels may be causal agents in the increase in diabetes and pre-diabetes. Other research supports this. This diabetes case study demonstrates that radiation from wireless technology can have profound effects on the ability of insulin to act in the body and, over time, on insulin sensitivity. Similar effects resulting from exposure to "dirty" electricity are also discussed. Furthermore, it is important for physicians to be aware of the potential for severe adverse effects to result from exposure to radiation from wireless technology, also discussed in the case study. This is not the only study showing that RF exposure can have profound endocrine effects in humans.  Other studies have shown effects on thyroid hormones, TSH, ACTH, cortisol, prolactin in females, and testosterone in males.  Changes in adrenaline, noradrenaline, dopamine, and phenylethylamine levels were found in response to an increase in ambient RF levels.  A mouse study found changes in cardiac function and structure can be caused by exposure to radiation from wireless technology, probably at least in part due to changes in electrolyte balance, since that same study found that renin levels were elevated.  This would be consistent with RF exposure affecting voltage-gated ion channels and with findings in humans that RF exposure causes cardiac arrhythmia.   A method for quickly setting an initial RF standard that would be protective against many of the biologically-based RF effects has been proposed in in a paper by Dr. Pall (https://www.ncbi.nlm.nih.gov/pubmed/25879308). It is based on preventing the effects on the voltage-gated calcium ion channels.