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This page is a summary of: Defects in mTR stability and telomerase activity produced by the Dkc1 A353V mutation in dyskeratosis congenita are rescued by a peptide from the dyskerin TruB domain, Clinical & Translational Oncology, July 2012, Springer Science + Business Media,
DOI: 10.1007/s12094-012-0865-4.
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