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  1. Ras inhibitor CAPRI enables neutrophil-like cells to chemotax through a higher-concentration range of gradients
  2. Filling GAPs in G protein- coupled receptor (GPCR)-mediated Ras adaptation and chemotaxis
  3. GPCR-controlled membrane recruitment of negative regulator C2GAP1 locally inhibits Ras signaling for adaptation and long-range chemotaxis