All Stories

  1. NOX-induced oxidative stress is a primary trigger of major neurodegenerative disorders

    Article • Progress in Neurobiology, December 2023, Elsevier

    Dr Misha Zilberter

  2. Neuronal APOE4 removal protects against tau-mediated gliosis, neurodegeneration and myelin deficits

    Article • Nature Aging, February 2023, Springer Science + Business Media

    Dr Misha Zilberter

  3. Author Correction: Experimental and real-world evidence supporting the computational repurposing of bumetanide for APOE4-related Alzheimer’s disease

    Article • Nature Aging, November 2021, Springer Science + Business Media

    Dr Misha Zilberter

  4. Experimental and real-world evidence supporting the computational repurposing of bumetanide for APOE4-related Alzheimer’s disease

    Article • Nature Aging, October 2021, Springer Science + Business Media

    Dr Misha Zilberter

  5. Aβ initiates brain hypometabolism, network dysfunction and behavioral abnormalities via NOX2-induced oxidative stress in mice

    Article • Communications Biology, September 2021, Springer Science + Business Media

    Dr Misha Zilberter

  6. Adult trkB Signaling in Parvalbumin Interneurons is Essential to Prefrontal Network Dynamics

    Article • Journal of Neuroscience, February 2021, Society for Neuroscience

    Dr Misha Zilberter

  7. Dentate Gyrus and CA3 GABAergic Interneurons Bidirectionally Modulate Signatures of Internal and External Drive to CA1

    Article • January 2021, Cold Spring Harbor Laboratory Press

    Dr Misha Zilberter

  8. Aβ-induced NOX2 activation underlies oxidative stress leading to brain hypometabolism and hyperactivity in Alzheimer's disease

    Article • August 2020, Cold Spring Harbor Laboratory Press

    Dr Misha Zilberter

  9. HCN channel activity balances quiescence and proliferation in neural stem cells and is a selective target for neuroprotection during cancer treatment.

    Article • Molecular Cancer Research, July 2020, American Association for Cancer Research (AACR)

    Dr Misha Zilberter

  10. In Vivo Chimeric Alzheimer’s Disease Modeling of Apolipoprotein E4 Toxicity in Human Neurons

    Article • Cell Reports, July 2020, Elsevier

    Dr Misha Zilberter, Emily A Jones

  11. A critical role for trkB signaling in the adult function of parvalbumin interneurons and prefrontal network dynamics

    Article • June 2020, Cold Spring Harbor Laboratory Press

    Dr Misha Zilberter

  12. Seizure-induced reduction in glucose utilization promotes brain hypometabolism during epileptogenesis

    Article • Neurobiology of Disease, April 2018, Elsevier

    Dr Misha Zilberter

  13. Metabolic correction by pyruvate halts acquired epilepsy in multiple rodent models

    Article • Neurobiology of Disease, October 2017, Elsevier

    Dr Misha Zilberter

  14. The vicious circle of hypometabolism in neurodegenerative diseases: Ways and mechanisms of metabolic correction

    Article • Journal of Neuroscience Research, May 2017, Wiley

    Dr Misha Zilberter

  15. Chronic inhibition of brain glycolysis initiates epileptogenesis

    Article • Journal of Neuroscience Research, February 2017, Wiley

    Dr Misha Zilberter

  16. Reality of Inhibitory GABA in Neonatal Brain: Time to Rewrite the Textbooks?

    Article • Journal of Neuroscience, October 2016, Society for Neuroscience

    Dr Misha Zilberter

  17. Triple-target treatment for Alzheimer’s: Correcting hypometabolism, oxidative stress, and neuroinflammation

    Article • Alzheimer s & Dementia, July 2015, Elsevier

    Dr Misha Zilberter

  18. Chemically defined, albumin-free human cardiomyocyte generation

    Article • Nature Chemical Biology, June 2015, Nature

    Dr Misha Zilberter

  19. Amyloid- -Induced Action Potential Desynchronization and Degradation of Hippocampal Gamma Oscillations Is Prevented by Interference with Peptide Conformation Change and Aggregation

    Article • Journal of Neuroscience, August 2014, Society for Neuroscience

    Dr Misha Zilberter

  20. Glycolysis and Oxidative Phosphorylation in Neurons and Astrocytes during Network Activity in Hippocampal Slices

    Article • Journal of Cerebral Blood Flow & Metabolism, December 2013, SAGE Publications

    Dr Misha Zilberter

  21. Dietary energy substrates reverse early neuronal hyperactivity in a mouse model of Alzheimer's disease

    Article • Journal of Neurochemistry, January 2013, Wiley

    Dr Misha Zilberter

  22. Molecular reorganization of endocannabinoid signalling in Alzheimer's disease

    Article • Brain, April 2011, Oxford University Press (OUP)

    Dr Misha Zilberter

  23. En masse in vitro functional profiling of the axonal mechanosensitivity of sensory neurons

    Article • Proceedings of the National Academy of Sciences, August 2010, Proceedings of the National Academy of Sciences

    Dr Misha Zilberter

  24. Secretagogin is a Ca 2+ -binding protein specifying subpopulations of telencephalic neurons

    Article • Proceedings of the National Academy of Sciences, December 2009, Proceedings of the National Academy of Sciences

    Dr Misha Zilberter

  25. beta-amyloid triggers neuronal hyper-activation and results in Alzheimer's-related epilepsy

    Article • Journal of Neuroscience, March 2009, Society for Neuroscience

    Dr Misha Zilberter

  26. Input Specificity and Dependence of Spike Timing-Dependent Plasticity on Preceding Postsynaptic Activity at Unitary Connections between Neocortical Layer 2/3 Pyramidal Cells

    Article • Cerebral Cortex, February 2009, Oxford University Press (OUP)

    Dr Misha Zilberter

  27. System A Transporter SAT2 Mediates Replenishment of Dendritic Glutamate Pools Controlling Retrograde Signaling by Glutamate

    Article • Cerebral Cortex, October 2008, Oxford University Press (OUP)

    Dr Misha Zilberter

  28. GABAergic control of backpropagating action potentials in striatal medium spiny neurons

    Article • BMC Neuroscience, January 2008, Springer Science + Business Media

    Dr Misha Zilberter

  29. Non-fibrillar β-amyloid abates spike-timing-dependent synaptic potentiation at excitatory synapses in layer 2/3 of the neocortex by targeting postsynaptic AMPA receptors

    Article • European Journal of Neuroscience, April 2006, Wiley

    Dr Misha Zilberter