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  1. Candidalysin Is Required for Neutrophil Recruitment and Virulence During Systemic Candida albicans Infection
  2. EphA2 Is a Neutrophil Receptor for Candida albicans that Stimulates Antifungal Activity during Oropharyngeal Infection
  3. Selection of Candida albicans trisomy during oropharyngeal infection results in a commensal-like phenotype
  4. Candida albicans at Host Barrier Sites: Pattern Recognition Receptors and Beyond
  5. Selection of Candida albicans Trisomy during Oropharyngeal Infection Results in a Commensal-Like Phenotype
  6. Inhibition of EGFR Signaling Protects from Mucormycosis
  7. Author Correction: EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  8. The Hyr1 protein from the fungus Candida albicans is a cross kingdom immunotherapeutic target for Acinetobacter bacterial infection
  9. Candida albicans White-Opaque Switching Influences Virulence but Not Mating during Oropharyngeal Candidiasis
  10. Publisher Correction: EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  11. EphA2 is an epithelial cell pattern recognition receptor for fungal β-glucans
  12. Innate Immunity to Mucosal Candida Infections
  13. The Aryl Hydrocarbon Receptor Governs Epithelial Cell Invasion during Oropharyngeal Candidiasis
  14. Oropharyngeal Candidiasis: Fungal Invasion and Epithelial Cell Responses
  15. AMPlified Defense: Antimicrobial Peptides During Candida albicans Infection
  16. Aspergillus fumigatus CalA binds to integrin α5β1 and mediates host cell invasion
  17. Candida albicans responds to glycostructure damage by Ace2‐mediated feedback regulation of Cek1 signaling
  18. Domains of CaMsb2
  19. Candida and AMPs
  20. C. albicans Mucin Msb2
  21. Msb2 Shedding