All Stories

  1. The origin of NMDA receptor hypofunction in schizophrenia
  2. AMPA Receptor Activation–Independent Antidepressant Actions of Ketamine Metabolite (S)-Norketamine
  3. Schizophrenia-Like Dopamine Release Abnormalities in a Mouse Model of NMDA Receptor Hypofunction
  4. Neuropsychiatric Phenotypes Produced by GABA Reduction in Mouse Cortex and Hippocampus
  5. Spatial and temporal boundaries of NMDA receptor hypofunction leading to schizophrenia
  6. Electrophysiological evidence for defective fast-spiking GABAergic neurones in a schizophrenia model
  7. Cortical GluN2B deletion attenuates punished suppression of food reward-seeking
  8. NMDA receptor subunits and associated signaling molecules mediating antidepressant-related effects of NMDA-GluN2B antagonism
  9. Impaired discrimination learning in interneuronal NMDAR-GluN2B mutant mice
  10. Brain state-dependent abnormal LFP activity in the auditory cortex of a schizophrenia mouse model
  11. Identification of Transcription Factors for Lineage-Specific ESC Differentiation
  12. Dysregulation of the axonal trafficking of nuclear-encoded mitochondrial mRNA alters neuronal mitochondrial activity and mouse behavior
  13. GluN2B in corticostriatal circuits governs choice learning and choice shifting
  14. Social Isolation Exacerbates Schizophrenia-Like Phenotypes via Oxidative Stress in Cortical Interneurons
  15. Contribution of NMDA Receptor Hypofunction in Prefrontal and Cortical Excitatory Neurons to Schizophrenia-Like Phenotypes
  16. Convergence of genetic and environmental factors on parvalbumin-positive interneurons in schizophrenia
  17. Hilar mossy cell circuitry controlling dentate granule cell excitability
  18. Preferential inactivation of Scn1a in parvalbumin interneurons increases seizure susceptibility
  19. Hilar Mossy Cell Degeneration Causes Transient Dentate Granule Cell Hyperexcitability and Impaired Pattern Separation
  20. GABAergic interneuron origin of schizophrenia pathophysiology
  21. Acute d-serine treatment produces antidepressant-like effects in rodents
  22. Chronic stress-induced hippocampal dendritic retraction requires CA3 NMDA receptors
  23. Genetically Engineered Mice for Schizophrenia Research
  24. Loss of GluN2B-Containing NMDA Receptors in CA1 Hippocampus and Cortex Impairs Long-Term Depression, Reduces Dendritic Spine Density, and Disrupts Learning
  25. eIF2  Phosphorylation-Dependent Translation in CA1 Pyramidal Cells Impairs Hippocampal Memory Consolidation without Affecting General Translation
  26. Postnatal NMDA receptor ablation in corticolimbic interneurons confers schizophrenia-like phenotypes
  27. Lack of kainic acid-induced gamma oscillations predicts subsequent CA1 excitotoxic cell death
  28. Chemically Inducible Inactivation of Protein Synthesis in Genetically Targeted Neurons
  29. Notch1 Signaling in Pyramidal Neurons Regulates Synaptic Connectivity and Experience-Dependent Modifications of Acuity in the Visual Cortex
  30. CA3 NMDA receptors are crucial for rapid and automatic representation of context memory
  31. Inducible and Cell-Type Restricted Manipulation in the Entorhinal Cortex
  32. Hippocampal CA3 NMDA Receptors Are Crucial for Adaptive Timing of Trace Eyeblink Conditioned Response
  33. Erratum
  34. NMDA Receptor-Dependent Ocular Dominance Plasticity in Adult Visual Cortex
  35. Genetic neuroscience of mammalian learning and memory
  36. Hippocampal CA3 NMDA Receptors Are Crucial for Memory Acquisition of One-Time Experience
  37. Essential function of α-calcium/calmodulin-dependent protein kinase II in neurotransmitter release at a glutamatergic central synapse
  38. Requirement for Hippocampal CA3 NMDA Receptors in Associative Memory Recall
  39. Antibody specific for phosphorylated AMPA-type glutamate receptors at GluR2 Ser-696
  40. Transient and persistent phosphorylation of AMPA-type glutamate receptor subunits in cerebellar Purkinje cells
  41. Immunohistochemical localization of protein phosphatase isoforms in the rat cerebellum
  42. On crucial roles of hippocampal NMDA receptors in acquisition and recall of associative memory