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  1. A role for Ral GTPase‐activating protein subunit β in mitotic regulation
  2. The bacterial pathogen Listeria monocytogenes and the interferon family: type I, type II and type III interferons
  3. Structural Basis for the Inhibition of the Chromatin Repressor BAHD1 by the Bacterial Nucleomodulin LntA
  4. Nuclear microbiology—bacterial assault on the nucleolus
  5. Murinization of Internalin Extends Its Receptor Repertoire, Altering Listeria monocytogenes Cell Tropism and Host Responses
  6. 1257 THE ACCUMULATION OF LIPID DROPLETS IN KUPFFER CELLS DISTURBS THEIR PHAGOCYTOSIS AND CLEARANCE FUNCTIONS
  7. Spatial positioning of cell wall-anchored virulence factors in Gram-positive bacteria
  8. Epigenetics and Bacterial Infections
  9. P128 Activation of type III interferon genes by pathogenic bacteria in infected epithelial cells and mouse placenta
  10. The New Microbiology: A conference at the Institut de France
  11. Activation of Type III Interferon Genes by Pathogenic Bacteria in Infected Epithelial Cells and Mouse Placenta
  12. When bacteria target the nucleus: the emerging family of nucleomodulins
  13. Bacteria tune interferon responses by playing with chromatin
  14. Regulated Shift from Helical to Polar Localization of Listeria monocytogenesCell Wall-Anchored Proteins
  15. A Bacterial Protein Targets the BAHD1 Chromatin Complex to Stimulate Type III Interferon Response
  16. The Stress-Induced Virulence Protein InlH Controls Interleukin-6 Production during Murine Listeriosis
  17. IlsA, A Unique Surface Protein of Bacillus cereus Required for Iron Acquisition from Heme, Hemoglobin and Ferritin
  18. Human BAHD1 promotes heterochromatic gene silencing
  19. Crystal Structure and Standardized Geometric Analysis of InlJ, a Listerial Virulence Factor and Leucine-Rich Repeat Protein with a Novel Cysteine Ladder
  20. Listeria monocytogenes internalins bind to the human intestinal mucin MUC2
  21. The Listeria monocytogenes Virulence Factor InlJ Is Specifically Expressed In Vivo and Behaves as an Adhesin
  22. Internalins: a complex family of leucine-rich repeat-containing proteins in Listeria monocytogenes
  23. Enterococcal Leucine-Rich Repeat-Containing Protein Involved in Virulence and Host Inflammatory Response
  24. Listeria monocytogenes Surface Proteins: from Genome Predictions to Function
  25. The Cell Wall of Listeria monocytogenes and its Role in Pathogenicity
  26. Listeria monocytogenes: a multifaceted model
  27. Species specificity of the Listeria monocytogenes InlB protein
  28. Identification of substrates of theListeria monocytogenes sortases A and B by a non-gel proteomic analysis
  29. LPXTG Protein InlJ, a Newly Identified Internalin Involved in Listeria monocytogenes Virulence
  30. Sorting sortases: a nomenclature proposal for the various sortases of Gram-positive bacteria
  31. WASP-related proteins, Abi1 and Ena/VASP are required for Listeria invasion induced by the Met receptor
  32. Analysis of theListeriacell wall proteome by two-dimensional nanoliquid chromatography coupled to mass spectrometry
  33. Sortase B, a New Class of Sortase in Listeria monocytogenes
  34. Inactivation of the srtA gene in Listeria monocytogenes inhibits anchoring of surface proteins and affects virulence
  35. A role for cofilin and LIM kinase in Listeria -induced phagocytosis
  36. The use of host cell machinery in the pathogenesis of Listeria monocytogenes
  37. The invasion protein InlB from Listeria monocytogenes activates PLC-gamma1 downstream from PI 3-kinase
  38. Interaction between the protein InlB of Listeria monocytogenes and lipoteichoic acid: a novel mechanism of protein association at the surface of Gram‐positive bacteria
  39. uvrD mutations enhance tandem repeat deletion in the Escherichia coli chromosome via SOS induction of the RecF recombination pathway
  40. InlB: an invasion protein of Listeria monocytogenes with a novel type of surface association
  41. Isolation of a dnaE mutation which enhances RecA‐independent homologous recombination in the Escherichia coli chromosome
  42. Deletions at stalled replication forks occur by two different pathways
  43. Competition between Parental and Recombinant Plasmids Affects the Measure of Recombination Frequencies
  44. When replication forks stop
  45. Mechanisms of illegitimate recombination